Daisy is due March 25, the same day my first grandchild is due. Daisy is showing signs of calving, her udder is beginning to swell with milk and her vulva is loosening up. Tomorrow is the full moon and we are going to also have a Lunar Eclipse.... exciting days ahead for us for sure!
Daisy is due on March 25 for her second calf. She started showing signs of calving last night and today I put her out on the back pasture. Spring came early so there is plenty of fresh new grass for her to graze on. We are very excited to welcome this little one to our ranch.
Right now I need to go and clean her pen and put out fresh bedding, just in case!
Our ultimate dream is to be able to offer a beautiful mid sized mini cow that is naturally polled with A2/A2 milk.
Naturally polled simply means that the cows will not have horns. Cows naturally do have horns but these days most people simply prefer that they don't. So far all the cows we have on our ranch carry at least one polled gene. For more information on what it means to be polled click here.
A2/A2 is a somewhat new thing that measures the beta-casein in a cows milk. It is considered safe and nutritious and has no known negative effects on human health. Sometime in the past few thousand years, a natural mutation occurred in some European dairy herds that changed the beta-casein they produced. The gene encoding beta-casein was switched from proline to histidine. This new kind of beta-casein that was created is known as A1 beta-casein, and is generally more common in many of the big black-and-white cow breeds of European descent such as the Holstein and Friesian. Due to their size, milk production, and demeanor, these breeds of cow are used to produce the vast majority of Northern Europe and America’s milk.
Each cow carries two copies of the gene encoding beta-casein, with a genotype of A1/A1, A1/A2, or A2/A2. Neither the A1 nor A2 trait appears to be dominant, which means that the milk produced by an A1/A2 cow will likely contain equal proportions of A1 and A2 beta-casein. A1/A1 cows will obviously only produce A1 beta-casein, just as A2/A2 cows will only produce A2 beta-casein. Northern European black-and-white breeds such as Friesian Holstein usually carry A1 and A2 alleles in equal proportion. Jersey cows and other Southern European breeds probably have about 1/3 A1 and 2/3 A2 genetics. Guernsey cows generally have about 10% A1 and 90% A2 genetics.
The cause for concern with milk containing A1 beta-casein is that the 67th amino acid switch from proline to histidine readily allows a digestive enzyme to cut out a 7 amino acid segment of the protein immediately adjacent to that histidine. When proline is present in that location (as it is in A2 beta-casein), that same segment is either not separated at all or the separation occurs at a very low rate. The 7 amino acid segment that is separated from A1 beta casein is known as beta-casomorphin-7, often abbreviated as BCM-7.
BCM-7 is the real “devil” in A1 milk for a number of reasons. It is an exogenous (doesn’t naturally occur within the human body) opioid that interacts with the human digestive system, internal organs, and brainstem. While no direct causal relationships have been demonstrated between BCM-7 and these diseases due to a wide range of contributing factors for each illness, BCM-7 has been linked to type 1 diabetes, heart disease, autism, and other serious non-communicable diseases as well.
It takes roughly two and a half years for a newborn calf to grow up, have a calf of its own, and begin producing milk, so having a completely A2/A2 herd occurs over a multiple-year time frame. Assuming the average cow is already 50% A2, but this is our goal.
For more information on A2/A2 click here.